Pulse Brain · Growing Health Evidence Index
Peer-reviewed

Genome-wide analyses of ADHD identify 27 risk loci, refine the genetic architecture and implicate several cognitive domains

Ditte Demontis, G. Bragi Walters, Georgios Athanasiadis, Raymond K. Walters, Karen Therrien, Trine Tollerup Nielsen, Leila Farajzadeh, Georgios Voloudakis, Jaroslav Bendl, Biau Zeng, Wen Zhang, Jakob Grove, Thomas D. Als, Jinjie Duan, F. Kyle Satterstrom, Jonas Bybjerg‐Grauholm, Marie Bækved-Hansen, Ólafur Ó. Guðmundsson, Sigurður H. Magnússon, Gísli Baldursson, Katrín Davíðsdóttir, Gyða S. Haraldsdóttir, Esben Agerbo, Gabriel E. Hoffman, Søren Dalsgaard, Joanna Martin, Marta Ribasès, Dorret I. Boomsma, María Soler Artigas, Nina Roth Mota, Daniel P. Howrigan, Sarah E. Medland, Tetyana Zayats, Veera M. Rajagopal, Alexandra Havdahl, Alysa E. Doyle, Andreas Reif, Anita Thapar, Bru Cormand, Calwing Liao, Christie L. Burton, Claiton H.D. Bau, Diego Luiz Rovaris, Edmund Sonuga‐Barke, Elizabeth C. Corfield, Eugênio H. Grevet, Henrik Larsson, Ian R. Gizer, Irwin D. Waldman, Isabell Brikell, Jan Haavik, Jennifer Crosbie, James J. McGough, Jonna Kuntsi, Joseph Glessner, K. Langley, Klaus‐Peter Lesch, Luís Augusto Rohde, Mara Helena Hutz, Marieke Klein, Mark A. Bellgrove, Martin Tesli, Michael O’Donovan, Ole A. Andreassen, Patrick W. L. Leung, Pedro Mário Pan, Ridha Joober, Russell Schachar, Sandra K. Loo, Stephanie H. Witt, Ted Reichborn‐Kjennerud, Tobias Banaschewski, Ziarih Hawi, Mark J. Daly, Ole Mors, Merete Nordentoft, Ole Mors, David M. Hougaard, Preben Bo Mortensen, Mark J. Daly, Stephen V. Faraone, Hreinn Stefánsson, Panos Roussos, Barbara Franke, Thomas Werge, Benjamin M. Neale, Kāri Stefánsson, Anders D. Børglum

Nature Genetics · 2023

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Summary

Attention-deficit hyperactivity disorder (ADHD) is a prevalent neurodevelopmental disorder with a major genetic component. Here, we present a genome-wide association study meta-analysis of ADHD comprising 38,691 individuals with ADHD and 186,843 controls. We identified 27 genome-wide significant loci, highlighting 76 potential risk genes enriched among genes expressed particularly in early brain development. Overall, ADHD genetic risk was associated with several brain-specific neuronal subtypes and midbrain dopaminergic neurons. In exome-sequencing data from 17,896 individuals, we identified an increased load of rare protein-truncating variants in ADHD for a set of risk genes enriched with probable causal common variants, potentially implicating SORCS3 in ADHD by both common and rare varia

Source type
Peer-reviewed study
DOI
10.1038/s41588-022-01285-8
Catalogue ID
SNmois81sf-1i030t
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