Pulse Brain · Growing Health Evidence Index
Tier 1 — Meta-analysis / systematic reviewPeer-reviewed

An atlas of genetic scores to predict multi-omic traits

Yu Xu, Scott C. Ritchie, Yujian Liang, Paul R. H. J. Timmers, Maik Pietzner, Loïc Lannelongue, Samuel A. Lambert, Usman A. Tahir, Sebastian May-Wilson, Carles Foguet, Åsa Johansson, Praveen Surendran, Artika P. Nath, Elodie Persyn, James E. Peters, Clare Oliver‐Williams, Shuliang Deng, Bram P. Prins, Jian’an Luan, Lorenzo Bomba, Nicole Soranzo, Emanuele Di Angelantonio, Nicola Pirastu, E Shyong Tai, Rob M. van Dam, Helen Parkinson, Emma E. Davenport, Dirk S. Paul, Christopher Yau, Robert E. Gerszten, Anders Mälarstig, John Danesh, Xueling Sim, Claudia Langenberg, James F. Wilson, Adam S. Butterworth, Michael Inouye

Nature · 2023

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Summary

This Nature publication presents a comprehensive atlas of genetic scores designed to predict multiple omic traits—including circulating proteins, metabolites, and lipids—across large populations. Through phenome-wide scanning, the authors identify novel disease associations and elucidate genetic mechanisms underlying metabolic pathways and disease processes, exemplified by JAK-STAT signalling in coronary atherosclerosis. The work culminates in a publicly accessible portal (omicspred.org) to democratise access to these polygenic scores and support future multi-omic prediction research.

Regional applicability

These genetic prediction tools and disease associations may be applicable to UK clinical and epidemiological research, particularly within NHS Biobank and other population cohorts. However, the transferability of polygenic scores across ancestry groups remains a consideration for equitable implementation in diverse UK populations.

Key measures

Polygenic risk scores; multi-omic trait predictions (proteins, metabolites, lipids); phenome-wide association scan results; disease associations; pathway-level biological mechanisms

Outcomes reported

The study developed and validated an atlas of genetic scores to predict multi-omic traits (proteins, metabolites, lipids) and conducted a phenome-wide scan to identify disease associations. The research identified biological insights regarding genetic mechanisms in metabolism and canonical pathway associations with disease, including JAK-STAT signalling and coronary atherosclerosis.

Theme
Measurement & metrics
Subject
Measurement methods & nutrient profiling
Study type
Research
Study design
Meta-analysis
Source type
Peer-reviewed study
Status
Published
Geography
International
System type
Human clinical
DOI
10.1038/s41586-023-05844-9
Catalogue ID
SNmoj1yirq-dd2gs8

Topic tags

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