Pulse Brain · Growing Health Evidence Index
Tier 3 — Observational / field trialPeer-reviewed

Genetic Variants in PHACTR1 & LPL Mediate Restenosis Risk in Coronary Artery Patients

Cynthia Al Hageh, Stéphanie Chacar, Thenmozhi Venkatachalam, Dominique Gauguier, Antoine Abchee, Elie Chammas, Hamdan Hamdan, Siobhán O’Sullivan, Pierre Zalloua, Moni Nader

Vascular Health and Risk Management · 2023

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Summary

This case-control genetic study of 5,242 coronary artery disease patients (2,099 genotyped) identified two single-nucleotide polymorphisms associated with restenosis risk following revascularisation. The PHACTR1 rs9349379 variant showed heightened association in women and diabetic patients, whilst the LPL rs264 variant was associated with increased restenosis risk in men. Dyslipidemia emerged as the dominant modifiable metabolic risk factor, with an odds ratio of 2.14 overall and 2.32 in men.

Regional applicability

The study cohort geography is not explicitly specified in the abstract; without confirmation of United Kingdom or European population origin, the generalisability to United Kingdom coronary artery disease populations cannot be established. Findings may inform personalised risk stratification in UK cardiology if the cohort demographics align with local populations.

Key measures

Odds ratios for restenosis risk; genetic association analysis via PLINK 1.9; stratification by sex, diabetes status, hypertension, and dyslipidemia

Outcomes reported

The study identified genetic variants (rs9349379 in PHACTR1 and rs264 in LPL) associated with increased restenosis risk following coronary revascularisation, with sex- and diabetes-dependent associations. Dyslipidemia emerged as a major modifiable risk factor for restenosis, particularly in men.

Theme
Nutrition & health
Subject
Out of scope / non-food
Study type
Research
Study design
Case-control association study
Source type
Peer-reviewed study
Status
Published
System type
Human clinical
DOI
10.2147/vhrm.s394695
Catalogue ID
SNmpeyuosy-63hbqa

Topic tags

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